【問題】HMGB1 inflammation ?推薦回答

關於「HMGB1 inflammation」標籤,搜尋引擎有相關的訊息討論:

HMGB1 released from nociceptors mediates inflammation | PNAS。

2021年8月17日 · Syn-Cre/HMGB1fl/fl mice subjected to experimental collagen antibody–induced arthritis, a disease model in which nociceptor-dependent ...: tw | tw。

High mobility group box 1 (HMGB1): a pivotal regulator of ...。

2020年7月13日 · HMGB1 is also involved in the formation of the inflammatory bone marrow (BM) ... which is inhibited by glycyrrhizin (GL) [111].。

Protective Effect of Glycyrrhizin, a Direct HMGB1 Inhibitor, on Focal ...。

2014年3月4日 · GL alone or GL and rHMGB1 were administered intravenously at the time of reperfusion. Serum levels of HMGB1 and inflammatory mediators were ...: tw | tw。

Protective effect of glycyrrhizin, a direct HMGB1 inhibitor, on post ...。

2021年8月2日 · After excretion from the cell, HMGB1 acts as a pro-inflammatory mediator ... Abbreviation: PC-AKI, post-contrast acute kidney injury; GL, ...。

HMGB1 Protein Does Not Mediate the Inflammatory Response in ...。

HMGB1 is beneficial for spontaneous spinal cord regeneration by eliciting negligible inflammation and promoting oligodendrocyte migration. Significance: HMGB1 ...。

HMGB1 Induces an Inflammatory Response in the Chorioamniotic ...。

Abstract. Spontaneous preterm labor occurs in two subsets of patients with sterile intra-amniotic inflammation, a process induced by alarmins such as ...。

Multiple Sclerosis and Neuroimmunology - Frontiers。

2018年7月2日 · The inflammatory mediator high-mobility group box 1 (HMGB1) plays a ... GL is a natural anti-inflammatory product compared to anti-HMGB1 ...。

HMGB1-Driven Inflammation and Intimal Hyperplasia After Arterial ...。

2015年10月29日 · HMGB1-Driven Inflammation and Intimal Hyperplasia After Arterial Injury ... Fokkema M, den Hartog AG, Bots ML, van der Tweel I, Moll FL, ...。

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HMGB1 could restrict 1,3-β-glucan induced mice lung inflammation ...。

Silencing HMGB1 aggravated inflammation by restricting epithelial cells autophagy. ... N. Marcussen, R. Jounblat, S. Chamat, U. Holmskov, G.L. Sorensen.


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